CXXC5 orchestrates Stat3/Erk/Akt signaling networks to modulate P. gingivalis-elicited autophagy in cementoblasts.
第一作者:
Li,Ma
第一单位:
The State Key Laboratory Breeding Base of Basic Science of Stomatology (Hubei-MOST KLOS) & Key Laboratory of Oral Biomedicine Ministry of Education (KLOBME), School & Hospital of Stomatology, Wuhan University, Wuhan, China.
作者:
主题词
动物(Animals);自噬(Autophagy);细菌学技术(Bacteriological Techniques);细胞培养技术(Cell Culture Techniques);细胞系(Cell Line);DNA结合蛋白质类(DNA-Binding Proteins);牙骨质(Dental Cementum);减量调节(Down-Regulation);基因表达调控(Gene Expression Regulation);MAP激酶信号系统(MAP Kinase Signaling System);小鼠(Mice);龈紫单胞菌(Porphyromonas gingivalis);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);STAT3转录因子(STAT3 Transcription Factor);转录因子(Transcription Factors)
DOI
10.1016/j.bbamcr.2020.118923
PMID
33285176
发布时间
2021-07-16
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Biochimica et biophysica acta. Molecular cell research
2021年1868卷3期
118923页
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