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首页> Life sciences> Attenuation of chronic arsenic neurotoxicity via melatonin in male offspring of maternal rats exposed to arsenic during conception: Involvement of oxidative DNA damage and inflammatory signaling cascades.

Attenuation of chronic arsenic neurotoxicity via melatonin in male offspring of maternal rats exposed to arsenic during conception: Involvement of oxidative DNA damage and inflammatory signaling cascades.

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作者： Naseh,Abdollahzade ^[1] ; Shirin,Babri ^[2] ; Maryam,Majidinia ^[3]

作者单位： Drug Applied Research Center, Tabriz University of Medical Sciences, Tabriz, Iran; Department of Physiology, School of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran. ^[1] Drug Applied Research Center, Tabriz University of Medical Sciences, Tabriz, Iran; Department of Physiology, School of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran. Electronic address: babris@tbzmed.ac.ir. ^[2] Solid Tumor Research Center, Urmia University of Medical Sciences, Urmia, Iran. Electronic address: majidinia.m@umsu.ac.ir. ^[3]

关键词 Arsenic Melatonin Neurodegenerative Oxidative stress Pregnancy

主题词动物(Animals);抗氧化剂(Antioxidants);细胞凋亡(Apoptosis);砷(Arsenic);女(雌)性(Female);炎症(Inflammation);男(雄)性(Male);褪黑激素(Melatonin);神经保护药(Neuroprotective Agents);神经中毒综合征(Neurotoxicity Syndromes);氧化性应激(Oxidative Stress);妊娠(Pregnancy);大鼠(Rats);大鼠, Wistar(Rats, Wistar);信号传导(Signal Transduction)

DOI 10.1016/j.lfs.2020.118876

PMID 33310035

发布时间 2021-02-01

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