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首页> Frontiers in immunology> MicroRNA-106a Inhibits Autophagy Process and Antimicrobial Responses by Targeting ULK1, ATG7, and ATG16L1 During Mycobacterial Infection.

MicroRNA-106a Inhibits Autophagy Process and Antimicrobial Responses by Targeting ULK1, ATG7, and ATG16L1 During Mycobacterial Infection.

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第一作者： Kunmei,Liu

第一单位： Ningxia Key Laboratory of Clinical and Pathogenic Microbiology, General Hospital of Ningxia Medical University, Yinchuan, China.

作者： Kunmei,Liu ^[1] ; Dantong,Hong ^[2] ; Fan,Zhang ^[3] ; Xin,Li ^[3] ; Meng,He ^[3] ; Xuebo,Han ^[3] ; Guolin,Zhang ^[3] ; Guangxian,Xu ^[4] ; Nicola J,Stonehouse ^[1] ; Zhongjia,Jiang ^[3] ; Weijun,An ^[5] ; Le,Guo ^[3]

作者单位： Ningxia Key Laboratory of Clinical and Pathogenic Microbiology, General Hospital of Ningxia Medical University, Yinchuan, China. ^[1] Ningxia Key Laboratory of Cerebrocranial Diseases, Ningxia Medical University, Yinchuan, China. ^[2] Department of Medical Laboratory, School of Clinical Medicine, Ningxia Medical University, Yinchuan, China. ^[3] Suzhou Institute for Drug Control, Suzhou, China. ^[4] School of Molecular and Cellular Biology, Faculty of Biological Sciences, University of Leeds, Leeds, United Kingdom. ^[5] Department of Orthopaedics, General Hospital of Ningxia Medical University, Yinchuan, China. ^[6]

关键词 ATG16L1 ATG7 Mycobacterium tuberculosis ULK1 autophagy miR-106a

主题词自噬(Autophagy);基因表达调控(Gene Expression Regulation);HEK293细胞(HEK293 Cells);宿主与病原体相互作用(Host-Pathogen Interactions);人类(Humans);免疫, 先天(Immunity, Innate);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);巨噬细胞(Macrophages);微RNAs(MicroRNAs);微生物生存能力(Microbial Viability);结核分枝杆菌(Mycobacterium tuberculosis);信号传导(Signal Transduction);结核(Tuberculosis)

DOI 10.3389/fimmu.2020.610021

PMID 33505399

发布时间 2023-12-13

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Frontiers in immunology

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