论文期刊

取消

高级检索

检索历史清除

首页> Life sciences> BBG enhances OLT1177-induced NLRP3 inflammasome inactivation by targeting P2X7R/NLRP3 and MyD88/NF-κB signaling in DSS-induced colitis in rats.

BBG enhances OLT1177-induced NLRP3 inflammasome inactivation by targeting P2X7R/NLRP3 and MyD88/NF-κB signaling in DSS-induced colitis in rats.

导出 LinkOut

翻译打印收藏纠错

作者： Sameh,Saber ^[1] ; Mahmoud E,Youssef ^[2] ; Hossam,Sharaf ^[3] ; Noha A,Amin ^[4] ; Ruwyda,El-Shedody ^[3] ; Farah H,Aboutouk ^[3] ; Yumna Abd,El-Galeel ^[3] ; Amr,El-Hefnawy ^[3] ; Dina,Shabaka ^[3] ; Arwa,Khalifa ^[3] ; Renad A,Saleh ^[3] ; Donya,Osama ^[3] ; Ghada,El-Zoghby ^[3] ; Naglaa A,Gobba ^[5]

作者单位： Department of Pharmacology, Faculty of Pharmacy, Delta University for Science and Technology, Gamasa, Egypt. Electronic address: sameh.mohamed@deltauniv.edu.eg. ^[1] Department of Pharmacology, Faculty of Pharmacy, Delta University for Science and Technology, Gamasa, Egypt. ^[2] Department of Clinical Pharmacy, Faculty of Pharmacy, Delta University for Science and Technology, Gamasa, Egypt. ^[3] Department of Haematology, Theodor Bilharz Research Institute, Egypt. ^[4] Department of Pharmacology and Toxicology, College of Pharmacy, Misr University for Science and Technology, Egypt. ^[5]

关键词 BBG Dextran sodium sulfate MyD88/NF-κB OLT1177 P2X7R/NLRP3 Ulcerative colitis

主题词动物(Animals);半胱氨酸天冬氨酸蛋白酶1(Caspase 1);结肠炎(Colitis);结肠炎, 溃疡性(Colitis, Ulcerative);细胞因子类(Cytokines);硫酸葡聚糖(Dextran Sulfate);炎性体(Inflammasomes);白细胞介素18(Interleukin-18);白细胞介素1β(Interleukin-1beta);男(雄)性(Male);髓样分化因子88(Myeloid Differentiation Factor 88);NF-κB(NF-kappa B);腈类(Nitriles);大鼠(Rats);大鼠, Wistar(Rats, Wistar);受体, 嘌呤能P2X7(Receptors, Purinergic P2X7);品红类染料(Rosaniline Dyes);信号传导(Signal Transduction)

DOI 10.1016/j.lfs.2021.119123

PMID 33548287

发布时间 2021-03-16

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

Life sciences

119123页

相似文献

中文期刊
外文期刊
学位论文
会议论文

特别提示：本网站仅提供医学学术资源服务，不销售任何药品和器械，有关药品和器械的销售信息，请查阅其他网站。

客服热线：4000-115-888 转3 (周一至周五：8:00至17:00)
|
客服邮箱：yiyao@wanfangdata.com.cn

违法和不良信息举报电话：4000-115-888，举报邮箱：problem@wanfangdata.com.cn，举报专区

官方微信

万方医学小程序

关于我们网站地图知识产权

北京万方数据股份有限公司

万方数据电子出版社

京ICP证010071号

京公网安备11010802020237号

京ICP备08100800号-1

违法和不良信息举报电话：4000-115-888，举报邮箱：problem@wanfangdata.com.cn，举报专区

new翻译充值订阅收藏移动端

官方微信

万方医学小程序

调查问卷