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首页> Journal of Alzheimer's disease : JAD> DAla2-GIP-GLU-PAL Protects Against Cognitive Deficits and Pathology in APP/PS1 Mice by Inhibiting Neuroinflammation and Upregulating cAMP/PKA/CREB Signaling Pathways.

DAla2-GIP-GLU-PAL Protects Against Cognitive Deficits and Pathology in APP/PS1 Mice by Inhibiting Neuroinflammation and Upregulating cAMP/PKA/CREB Signaling Pathways.

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第一作者： Li,Yuan

第一单位： Department of Physiology, Changzhi Medical College, Changzhi, Shanxi, PR China.

作者： Li,Yuan ^[1] ; Jun,Zhang ^[2] ; Jun-Hong,Guo ^[2] ; Christian,Holscher ^[3] ; Jun-Ting,Yang ^[4] ; Mei-Na,Wu ^[2] ; Zhao-Jun,Wang ^[2] ; Hong-Yan,Cai ^[2] ; Ling-Na,Han ^[5] ; Hui,Shi ^[1] ; Yu-Fei,Han ^[2] ; Jin-Shun,Qi ^[2]

作者单位： Department of Physiology, Changzhi Medical College, Changzhi, Shanxi, PR China. ^[1] Department of Physiology, Key Laboratory of Cellular Physiology, Ministry of Education, Shanxi Medical University, Taiyuan, Shanxi, PR China. ^[2] Department of Neurology, First Hospital, Shanxi Medical University, Taiyuan, Shanxi, PR China. ^[3] Research and Experimental Center, Henan University of Chinese Medicine, Zhengzhou, Henan, PR China. ^[4] Department of Immunology and Microbiology, Shanxi Medical University, Taiyuan, Shanxi, PR China. ^[5]

关键词 Amyloid-β DAla2GIP-Glu-PAL cognitive behaviors long-term synaptic plasticity neuroinflammation

主题词阿尔茨海默病(Alzheimer Disease);淀粉样β肽类(Amyloid beta-Peptides);动物(Animals);认知(Cognition);疾病模型, 动物(Disease Models, Animal);肠抑胃肽(Gastric Inhibitory Polypeptide);海马(Hippocampus);长时程增强(Long-Term Potentiation);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);神经变性疾病(Neurodegenerative Diseases);神经元可塑性(Neuronal Plasticity);神经保护药(Neuroprotective Agents);斑块, 淀粉样(Plaque, Amyloid)

DOI 10.3233/JAD-201262

PMID 33579843

发布时间 2021-09-06

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Journal of Alzheimer's disease : JAD

Journal of Alzheimer's disease

2021年80卷2期

695-713页

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