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首页> International immunopharmacology> Glutaredoxin 2 protects cardiomyocytes from hypoxia/reoxygenation-induced injury by suppressing apoptosis, oxidative stress, and inflammation via enhancing Nrf2 signaling.

Glutaredoxin 2 protects cardiomyocytes from hypoxia/reoxygenation-induced injury by suppressing apoptosis, oxidative stress, and inflammation via enhancing Nrf2 signaling.

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作者： Chengliang,Li ^[1] ; Hong,Xin ^[2] ; Yingpeng,Shi ^[3] ; Jianjun,Mu ^[4]

作者单位： Department of Cardiology, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, China; Department of General Practice, The First Affiliated Hospital of Xi'an Medical University, Xi'an 710077, China. ^[1] Department of Cardiology, The First Affiliated Hospital of Xi'an Medical University, Xi'an 710077, China. ^[2] Department of General Practice, The First Affiliated Hospital of Xi'an Medical University, Xi'an 710077, China. ^[3] Department of Cardiology, The First Affiliated Hospital of Xi'an Jiaotong University, Xi'an 710061, China. Electronic address: mujianjunxjtu@163.com. ^[4]

关键词 Cardiomyocyte GRX2 Hypoxia/reoxygenation Nrf2 Oxidative stress

主题词动物(Animals);细胞凋亡(Apoptosis);细胞低氧(Cell Hypoxia);细胞系(Cell Line);细胞因子类(Cytokines);谷氧还蛋白质类(Glutaredoxins);炎症(Inflammation);小鼠(Mice);心肌再灌注损伤(Myocardial Reperfusion Injury);肌细胞, 心脏(Myocytes, Cardiac);NF-E2相关因子2(NF-E2-Related Factor 2);氧化性应激(Oxidative Stress);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction)

DOI 10.1016/j.intimp.2021.107428

PMID 33581580

发布时间 2021-06-22

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International immunopharmacology

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