Aging imparts cell-autonomous dysfunction to regulatory T cells during recovery from influenza pneumonia.

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作者： Luisa,Morales-Nebreda ^[1] ; Kathryn A,Helmin ^[1] ; Manuel A,Torres Acosta ^[1] ; Nikolay S,Markov ^[1] ; Jennifer Yuan-Shih,Hu ^[1] ; Anthony M,Joudi ^[1] ; Raul,Piseaux-Aillon ^[1] ; Hiam,Abdala-Valencia ^[1] ; Yuliya,Politanska ^[1] ; Benjamin D,Singer ^[1]

作者单位： Department of Medicine, Division of Pulmonary and Critical Care Medicine. ^[1] Department of Biochemistry and Molecular Genetics, and. ^[2] Simpson Querrey Institute for Epigenetics, Northwestern University Feinberg School of Medicine, Chicago, Illinois, USA. ^[3]

关键词 Aging T cells

主题词年龄因素(Age Factors);衰老(Aging);动物(Animals);人类(Humans);流感病毒A型(Influenza A virus);流感, 人(Influenza, Human);肺(Lung);小鼠, 近交C57BL(Mice, Inbred C57BL);肺炎, 病毒性(Pneumonia, Viral);T淋巴细胞, 调节性(T-Lymphocytes, Regulatory);小鼠(Mice)

DOI 10.1172/jci.insight.141690

PMID 33600379

发布时间 2024-02-26

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Aging imparts cell-autonomous dysfunction to regulatory T cells during recovery from influenza pneumonia.

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Aging imparts cell-autonomous dysfunction to regulatory T cells during recovery from influenza pneumonia.

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