Paclitaxel mitigates structural alterations and cardiac conduction system defects in a mouse model of Hutchinson-Gilford progeria syndrome.
第一作者:
Álvaro,Macías
第一单位:
Vascular Pathophysiology Area, Centro Nacional de Investigaciones Cardiovasculares Carlos III (CNIC), 28029 Madrid, Spain.
作者:
主题词
动作电位(Action Potentials);动物(Animals);抗心律失常药(Anti-Arrhythmia Agents);心律失常, 心性(Arrhythmias, Cardiac);细胞骨架(Cytoskeleton);疾病模型, 动物(Disease Models, Animal);兴奋收缩偶联(Excitation Contraction Coupling);女(雌)性(Female);疾病遗传易感性(Genetic Predisposition to Disease);心脏传导系统(Heart Conduction System);心率(Heart Rate);核纤层蛋白A型(Lamin Type A);男(雄)性(Male);小鼠, 突变型(Mice, Mutant Strains);突变(Mutation);肌细胞, 心脏(Myocytes, Cardiac);紫杉酚(Paclitaxel);早衰(Progeria);不应期, 电生理学(Refractory Period, Electrophysiological);猪(Swine);猪, 雏型(Swine, Miniature);微管蛋白(Tubulin);小鼠(Mice)
DOI
10.1093/cvr/cvab055
PMID
33624748
发布时间
2024-02-26
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Cardiovascular research
503-516页
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