Long QT Syndrome <i>KCNH2</i> Variant Induces hERG1a/1b Subunit Imbalance in Patient-Specific Induced Pluripotent Stem Cell-Derived Cardiomyocytes.

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作者： Li,Feng ^[1] ; Jianhua,Zhang ^[2] ; ChangHwan,Lee ^[1] ; Gina,Kim ^[3] ; Fang,Liu ^[1] ; Andrew J,Petersen ^[4] ; Evi,Lim ^[5] ; Corey L,Anderson ^[1] ; Kate M,Orland ^[1] ; Gail A,Robertson ^[1] ; Lee L,Eckhardt ^[4] ; Craig T,January ^[1] ; Timothy J,Kamp ^[1]

作者单位： Cellular and Molecular Arrhythmia Research Program, Division of Cardiovascular Medicine, Department of Medicine (L.F., J.Z., G.K., E.L., C.L.A., K.M.O., L.L.E., C.T.J., T.J.K.), University of Wisconsin-Madison. ^[1] Department of Cardiology, Beijing Anzhen Hospital, Capital Medical University, National Clinical Research Center for Cardiovascular Diseases, China (L.F.). ^[2] Department of Biological Sciences, University at Albany, State University of New York (C.L.). ^[3] Department of Neuroscience, Wisconsin Institutes for Medical Research (F.L., G.A.R.), University of Wisconsin-Madison. ^[4] Waisman Center (A.J.P.), University of Wisconsin-Madison. ^[5] Department of Cell and Regenerative Biology (T.J.K.), University of Wisconsin-Madison. ^[6]

关键词 electrophysiology ion channel long QT syndrome phenotype stem cell