Rapid initiation of cell cycle reentry processes protects neurons from amyloid-β toxicity.

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第一作者： Stefania,Ippati

第一单位： Dementia Research Centre, Department of Biomedical Sciences, Faculty of Medicine Health and Human Sciences, Macquarie University, Sydney, NSW 2109, Australia; stefania.ippati@hsr.it lars.ittner@mq.edu.au.

作者： Stefania,Ippati ^[1] ; Yuanyuan,Deng ^[2] ; Julia,van der Hoven ^[3] ; Celine,Heu ^[3] ; Annika,van Hummel ^[4] ; Sook Wern,Chua ^[3] ; Esmeralda,Paric ^[3] ; Gabriella,Chan ^[3] ; Astrid,Feiten ^[3] ; Thomas,Fath ^[3] ; Yazi D,Ke ^[3] ; Nikolas K,Haass ^[3] ; Lars M,Ittner ^[5]

作者单位： Dementia Research Centre, Department of Biomedical Sciences, Faculty of Medicine Health and Human Sciences, Macquarie University, Sydney, NSW 2109, Australia; stefania.ippati@hsr.it lars.ittner@mq.edu.au. ^[1] San Raffaele Scientific Institute, San Raffaele Hospital, 20132 Milan, Italy. ^[2] Dementia Research Centre, Department of Biomedical Sciences, Faculty of Medicine Health and Human Sciences, Macquarie University, Sydney, NSW 2109, Australia. ^[3] Biomedical Imaging Facility, Mark Wainwright Analytical Centre, University of New South Wales, Sydney NSW 2052, Australia. ^[4] The University of Queensland Diamantina Institute, The University of Queensland, Brisbane, QLD 4102, Australia. ^[5]

关键词 Alzheimer FUCCI cell cycle postmitotic neurons