MitoQ protects against hyperpermeability of endothelium barrier in acute lung injury via a Nrf2-dependent mechanism.

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作者： Mengyuan,Cen ^[1] ; Wei,Ouyang ^[1] ; Wanying,Zhang ^[1] ; Liping,Yang ^[1] ; Xiuhui,Lin ^[1] ; Min,Dai ^[1] ; Huiqun,Hu ^[1] ; Huifang,Tang ^[2] ; Hongyun,Liu ^[3] ; Jingyan,Xia ^[4] ; Feng,Xu ^[5]

作者单位： Department of Infectious Diseases, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310009, China. ^[1] Department of Pharmacology, School of Basic Medical Sciences, Zhejiang University, Hangzhou, Zhejiang 310058, China. ^[2] College of Animal Sciences, Zhejiang University, Hangzhou, Zhejiang 310058, China. ^[3] Department of Radiation Oncology, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310009, China. Electronic address: xiajingyan2018@zju.edu.cn. ^[4] Department of Infectious Diseases, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou, 310009, China. Electronic address: xufeng99@zju.edu.cn. ^[5]

关键词 ALI Apoptosis Endothelial cells MitoQ Nrf2

主题词急性肺损伤(Acute Lung Injury);动物(Animals);抗氧化剂(Antioxidants);内皮细胞(Endothelial Cells);内皮(Endothelium);脂多糖类(Lipopolysaccharides);小鼠(Mice);NF-E2相关因子2(NF-E2-Related Factor 2);信号传导(Signal Transduction)

DOI 10.1016/j.redox.2021.101936

PMID 33752110

发布时间 2022-05-31

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Redox biology

2021年41卷

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MitoQ protects against hyperpermeability of endothelium barrier in acute lung injury via a Nrf2-dependent mechanism.

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MitoQ protects against hyperpermeability of endothelium barrier in acute lung injury via a Nrf2-dependent mechanism.

Redox biology

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Redox biology

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