Loss of SMAD4 Is Sufficient to Promote Tumorigenesis in a Model of Dysplastic Barrett's Esophagus.

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作者： Jovana R,Gotovac ^[1] ; Tanjina,Kader ^[1] ; Julia V,Milne ^[1] ; Kenji M,Fujihara ^[1] ; Luis E,Lara-Gonzalez ^[1] ; Kylie L,Gorringe ^[1] ; Sangeetha N,Kalimuthu ^[2] ; Madawa W,Jayawardana ^[1] ; Cuong P,Duong ^[1] ; Wayne A,Phillips ^[3] ; Nicholas J,Clemons ^[4]

作者单位： Division of Cancer Research, Peter MacCallum Cancer Centre, Melbourne, Victoria, Australia; Sir Peter MacCallum Department of Oncology, Parkville, Victoria, Australia. ^[1] Anatomical Pathology, Laboratory Medicine Program, University Health Network, Toronto, Ontario, Canada; Laboratory Medicine and Pathobiology, University of Toronto, Toronto, Ontario, Canada. ^[2] Division of Cancer Research, Peter MacCallum Cancer Centre, Melbourne, Victoria, Australia; Sir Peter MacCallum Department of Oncology, Parkville, Victoria, Australia; Department of Surgery, St Vincent's Hospital, The University of Melbourne, Parkville, Victoria, Australia. ^[3] Division of Cancer Research, Peter MacCallum Cancer Centre, Melbourne, Victoria, Australia; Sir Peter MacCallum Department of Oncology, Parkville, Victoria, Australia. Electronic address: nicholas.clemons@petermac.org. ^[4]

关键词 Barrett’s Tumorigenesis Copy Number Alterations (CNA)Esophageal Adenocarcinoma (EAC)SMAD4 Loss

主题词动物(Animals);Barrett食管(Barrett Esophagus);碱基序列(Base Sequence);细胞系(Cell Line);减量调节(Down-Regulation);基因剂量(Gene Dosage);基因, 肿瘤抑制(Genes, Tumor Suppressor);人类(Humans);小鼠(Mice);肿瘤转移(Neoplasm Metastasis);癌基因(Oncogenes);主成分分析(Principal Component Analysis);信号传导(Signal Transduction);Smad4蛋白质(Smad4 Protein);转化生长因子β(Transforming Growth Factor beta);异种移植模型抗肿瘤试验(Xenograft Model Antitumor Assays)

DOI 10.1016/j.jcmgh.2021.03.008

PMID 33774196

发布时间 2023-09-20

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Cellular and molecular gastroenterology and hepatology

2021年12卷2期

689-713页

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Loss of SMAD4 Is Sufficient to Promote Tumorigenesis in a Model of Dysplastic Barrett's Esophagus.

Cellular and molecular gastroenterology and hepatology

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Cellular and molecular gastroenterology and hepatology

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Loss of SMAD4 Is Sufficient to Promote Tumorigenesis in a Model of Dysplastic Barrett's Esophagus.

Cellular and molecular gastroenterology and hepatology

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Cellular and molecular gastroenterology and hepatology

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