Connecting TDP-43 Pathology with Neuropathy.

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作者： Joseph R,Klim ^[1] ; Greta,Pintacuda ^[1] ; Leslie A,Nash ^[1] ; Irune,Guerra San Juan ^[2] ; Kevin,Eggan ^[3]

作者单位： Department of Stem Cell and Regenerative Biology, Department of Molecular and Cellular Biology, and Harvard Stem Cell Institute, Cambridge, MA 02138, USA; Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA. ^[1] Department of Stem Cell and Regenerative Biology, Department of Molecular and Cellular Biology, and Harvard Stem Cell Institute, Cambridge, MA 02138, USA; Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA; Department of Functional Genomics, Center for Neurogenomics and Cognitive Research, Vrije Universiteit Amsterdam, Amsterdam, 1081 HV, The Netherlands. ^[2] Department of Stem Cell and Regenerative Biology, Department of Molecular and Cellular Biology, and Harvard Stem Cell Institute, Cambridge, MA 02138, USA; Stanley Center for Psychiatric Research, Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA. Electronic address: eggan@mcb.harvard.edu. ^[3]

关键词 RNA binding protein SCG10 Stathmin2 amyotrophic lateral sclerosis (ALS)frontotemporal dementia (FTD)motor neuron disease

主题词肌萎缩侧索硬化(Amyotrophic Lateral Sclerosis);轴突(Axons);DNA结合蛋白质类(DNA-Binding Proteins);额颞叶痴呆(Frontotemporal Dementia);人类(Humans);突变(Mutation)

DOI 10.1016/j.tins.2021.02.008

PMID 33832769

发布时间 2022-05-31

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