Germacrone improves liver fibrosis by regulating the PI3K/AKT/mTOR signalling pathway.

导出 LinkOut

翻译打印收藏纠错

第一作者： De,Ji

第一单位： Department of Chinese Medicinal Processing, School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, China.;Molecular Pharmacology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.

作者： De,Ji ^[1] ; Qi,Zhao ^[2] ; Yuwen,Qin ^[2] ; Huangjin,Tong ^[3] ; Qiaohan,Wang ^[4] ; Mengting,Yu ^[4] ; Chunqin,Mao ^[4] ; Tulin,Lu ^[4] ; Jinchun,Qiu ^[5] ; Chengxi,Jiang ^[2]

作者单位： Department of Chinese Medicinal Processing, School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, China.;Molecular Pharmacology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China. ^[1] Molecular Pharmacology Research Center, School of Pharmaceutical Science, Wenzhou Medical University, Wenzhou, Zhejiang, China.;Department of Chinese Medicinal Resources, Biomedical Collaborative Innovation Center of Zhejiang, Wenzhou, China. ^[2] Department of Chinese Medicinal Processing, School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, China.;Department of Pharmacy, Affiliated Hospital of Integrated Traditional Chinese and Western Medicine, Nanjing University of Chinese Medicine, Nanjing, China. ^[3] Department of Chinese Medicinal Processing, School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing, China. ^[4] Department of Pharmacy, Children's Hospital of Nanjing Medical University, Nanjing, China. ^[5]

关键词 Germacrone PI3K/AKT/mTOR signalling pathway epithelial-mesenchymal transition hepatic stellate cell activation liver fibrosis proliferation