Impaired TRPV4-eNOS signaling in trabecular meshwork elevates intraocular pressure in glaucoma.

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作者： Pinkal D,Patel ^[1] ; Yen-Lin,Chen ^[2] ; Ramesh B,Kasetti ^[1] ; Prabhavathi,Maddineni ^[1] ; William,Mayhew ^[1] ; J Cameron,Millar ^[1] ; Dorette Z,Ellis ^[3] ; Swapnil K,Sonkusare ^[4] ; Gulab S,Zode ^[5]

作者单位： Department of Pharmacology and Neuroscience, North Texas Eye Research Institute, University of North Texas Health Science Center at Fort Worth, Fort Worth, TX 76107. ^[1] Robert M. Berne Cardiovascular Research Center, University of Virginia, Charlottesville, VA 22908. ^[2] Department of Pharmaceutical Sciences, North Texas Eye Research Institute, University of North Texas Health Science Center at Fort Worth, Fort Worth, TX 76107. ^[3] Robert M. Berne Cardiovascular Research Center, University of Virginia, Charlottesville, VA 22908; swapnil.sonkusare@virginia.edu gulab.zode@unthsc.edu. ^[4] Molecular Physiology and Biological Physics, University of Virginia School of Medicine, Charlottesville, VA 22908. ^[5] Department of Pharmacology and Neuroscience, North Texas Eye Research Institute, University of North Texas Health Science Center at Fort Worth, Fort Worth, TX 76107; swapnil.sonkusare@virginia.edu gulab.zode@unthsc.edu. ^[6]

关键词 TRPV4 fluid flow glaucoma nitric oxide trabecular meshwork