换一批
换一批Innate immunity to yeast prions: Btn2p and Cur1p curing of the [URE3] prion is prevented by 60S ribosomal protein deficiency or ubiquitin/proteasome system overactivity.
第一作者：
Evgeny E,Bezsonov
第一单位：
Laboratory of Biochemistry and Genetics, National Institute of Diabetes and Digestive and Kidney Diseases, National Institutes of Health, Bethesda, MD 20892-0830, USA.
作者：
主题词
氨基酸转运系统(Amino Acid Transport Systems);谷胱甘肽过氧化酶(Glutathione Peroxidase);HSP40热休克蛋白质类(HSP40 Heat-Shock Proteins);热休克蛋白质类(Heat-Shock Proteins);免疫, 先天(Immunity, Innate);分子伴侣(Molecular Chaperones);朊病毒(Prions);蛋白酶体内肽酶复合物(Proteasome Endopeptidase Complex);核糖体蛋白质类(Ribosomal Proteins);核糖体亚单位, 大, 真核细胞(Ribosome Subunits, Large, Eukaryotic);酿酒酵母菌(Saccharomyces cerevisiae);酿酒酵母蛋白质类(Saccharomyces cerevisiae Proteins);泛素蛋白连接酶类(Ubiquitin-Protein Ligases);泛素化(Ubiquitination)
DOI
10.1093/genetics/iyab013
PMID
33857305
发布时间
2022-02-04
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