High glucose protects cardiomyocytes against ischaemia/reperfusion injury by suppressing myocardiocyte apoptosis via circHIPK3/miR-29b/AKT3 signalling.

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第一作者： Nan,Cheng

第一单位： Department of Cardiovascular Surgery, Chinese PLA General Hospital, Beijing, China.

作者： Nan,Cheng ^[1] ; Chang,Jin ^[2] ; Ping,Jin ^[3] ; Dan,Zhu ^[4] ; Zuoxu,Hou ^[5]

作者单位： Department of Cardiovascular Surgery, Chinese PLA General Hospital, Beijing, China. ^[1] Medical Innovation Research Division, Research Center for Biomedical Engineering, Chinese PLA General Hospital, Beijing, China. ^[2] Department of Cardiovascular Surgery, The First Affiliated Hospital of Air Force Military Medical University, Xi'an, China. ^[3] Department of Arrhythmia, Qinghai Cardio-Cerebrovascular Hospital, Xi'ning, China. ^[4] Department of Thoracic Cardiovascular Surgery, General Hospital of Central Theater Command of the People's Liberation Army, Wuhan, China. ^[5]

关键词 AKT3 apoptosis circHIPK3 high glucose ischaemia/reperfusion miR-29b

DOI 10.1111/jcmm.16527

PMID 33951290

发布时间 2024-02-22

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Journal of cellular and molecular medicine

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High glucose protects cardiomyocytes against ischaemia/reperfusion injury by suppressing myocardiocyte apoptosis via circHIPK3/miR-29b/AKT3 signalling.

Journal of cellular and molecular medicine

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Journal of cellular and molecular medicine

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High glucose protects cardiomyocytes against ischaemia/reperfusion injury by suppressing myocardiocyte apoptosis via circHIPK3/miR-29b/AKT3 signalling.

Journal of cellular and molecular medicine

相似文献

Journal of cellular and molecular medicine

相关期刊

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