Gamma-Decanolactone Alters the Expression of GluN2B, A<sub>1</sub> Receptors, and COX-2 and Reduces DNA Damage in the PTZ-Induced Seizure Model After Subchronic Treatment in Mice.

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作者： Fernanda Marcelia,Dos Santos ^[1] ; Pricila Fernandes,Pflüger ^[1] ; Leticia,Lazzarotto ^[1] ; Mariana,Uczay ^[1] ; Wesley Roberto,de Aguida ^[1] ; Lisiane Santos,da Silva ^[2] ; Fernanda Brião Menezes,Boaretto ^[3] ; Jayne Torres,de Sousa ^[3] ; Jaqueline Nascimento,Picada ^[3] ; Iraci Lucena,da Silva Torres ^[2] ; Patrícia,Pereira ^[4]

作者单位： Laboratory of Neuropharmacology and Preclinical Toxicology, Health Basic Sciences Institute, Federal University of Rio Grande do Sul, Porto Alegre, RS, Brazil. ^[1] Laboratory of Pain Pharmacology and Neuromodulation: Pre-Clinical Research. Postgraduate Program in Medical Sciences, School of Medicine, Universidade Federal do Rio Grande do Sul, Porto Alegre, RS, Brazil. ^[2] Laboratory of Genetic Toxicology, Lutheran University of Brazil, Canoas, RS, Brazil. ^[3] Laboratory of Neuropharmacology and Preclinical Toxicology, Health Basic Sciences Institute, Federal University of Rio Grande do Sul, Porto Alegre, RS, Brazil. patriciapereira@ufrgs.br. ^[4] Laboratory of Neuropharmacology and Preclinical Toxicology, Department of Pharmacology, Institute of Basic Health Sciences, Federal University of Rio Grande do Sul, Sarmento Leite 500/305, Porto Alegre, RS, CEP 90050-170, Brazil. patriciapereira@ufrgs.br. ^[5]

关键词 Adenosine A1 receptor COX-2 Gamma-decanolactone GluN2B Pentylenetetrazole Seizure