miR-204 silencing reduces mitochondrial autophagy and ROS production in a murine AD model via the TRPML1-activated STAT3 pathway.

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作者： Lu,Zhang ^[1] ; Yu,Fang ^[2] ; Xinyu,Zhao ^[1] ; Yake,Zheng ^[1] ; Yunqing,Ma ^[1] ; Shuang,Li ^[1] ; Zhi,Huang ^[1] ; Lihao,Li ^[1]

作者单位： Department of Neurology, the First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, P.R. China. ^[1] ICU, The First Affiliated Hospital of Zhengzhou University, Zhengzhou 450052, P.R. China. ^[2]

关键词 Alzheimer’s disease Aβ1-42 hippocampal neurons microRNA-204 mitochondrial autophagy reactive oxygen species signal transducer and activator of transcription 3 transient receptor potential mucolipin-1

DOI 10.1016/j.omtn.2021.02.010

PMID 34026326

发布时间 2024-08-10

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Molecular therapy. Nucleic acids

2021年24卷

822-831页

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miR-204 silencing reduces mitochondrial autophagy and ROS production in a murine AD model via the TRPML1-activated STAT3 pathway.

Molecular therapy. Nucleic acids

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Molecular therapy. Nucleic acids

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miR-204 silencing reduces mitochondrial autophagy and ROS production in a murine AD model via the TRPML1-activated STAT3 pathway.

Molecular therapy. Nucleic acids

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Molecular therapy. Nucleic acids

相关期刊

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