Evaluating causality of cellular senescence in non-alcoholic fatty liver disease.

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第一作者： Abraham Stijn,Meijnikman

第一单位： Department of Internal and Vascular Medicine, Amsterdam University Medical Centers, location AMC, Amsterdam, the Netherlands.

作者： Abraham Stijn,Meijnikman ^[1] ; Hilde,Herrema ^[1] ; Torsten Pascal Marcel,Scheithauer ^[1] ; Jeffrey,Kroon ^[1] ; Max,Nieuwdorp ^[1] ; Albert Kornelis,Groen ^[1]

作者单位： Department of Internal and Vascular Medicine, Amsterdam University Medical Centers, location AMC, Amsterdam, the Netherlands. ^[1]

关键词 ATM, ataxia telangiectasia mutated C/EBPα, CCAAT- enhancer-binding protein CDK, cyclin dependent kinase DDR, DNA damage response FFAs, free fatty acids HCC, hepatocellular carcinoma IL-, interleukin KC, Kupffer cell LSEC, liver sinusoidal endothelial cell MCP1/CCL2, monocyte chemoattractant protein-1 MiDAS, mitochondrial dysfunction-associated senescence NAFL, non-alcoholic fatty liver NAFLD, non-alcoholic fatty liver disease NASH, non-alcoholic steatohepatitis ROS, reactive oxygen species Rb, retinoblastoma factor SA-β gal, senescence-associated beta-galactosidase SASP, senescence-associated secretory phenotype SCAP, senescence-associated antiapoptotic pathways TGFβ, transforming growth factor-β TNFα, tumour necrosis factor-α cellular senescence non-alcoholic fatty liver disease non-alcoholic steatohepatitis obesity qPCR, quantitative PCR senolytics

DOI 10.1016/j.jhepr.2021.100301

PMID 34113839

发布时间 2021-06-12

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JHEP reports

2021年3卷4期

100301页

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Evaluating causality of cellular senescence in non-alcoholic fatty liver disease.

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Evaluating causality of cellular senescence in non-alcoholic fatty liver disease.

JHEP reports

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