Overexpression of human BAG3<sup>P209L</sup> in mice causes restrictive cardiomyopathy.-论文-万方医学网

第一作者： Kenichi,Kimura

第一单位： Institute of Physiology I, Life and Brain Center, Medical Faculty, University of Bonn, Bonn, Germany.

作者： Kenichi,Kimura ^[1] ; Astrid,Ooms ^[1] ; Kathrin,Graf-Riesen ^[1] ; Maithreyan,Kuppusamy ^[2] ; Andreas,Unger ^[3] ; Julia,Schuld ^[4] ; Jan,Daerr ^[4] ; Achim,Lother ^[5] ; Caroline,Geisen ^[1] ; Lutz,Hein ^[5] ; Satoru,Takahashi ^[6] ; Guang,Li ^[7] ; Wilhelm,Röll ^[8] ; Wilhelm,Bloch ^[9] ; Peter F M,van der Ven ^[4] ; Wolfgang A,Linke ^[3] ; Sean M,Wu ^[7] ; Pitter F,Huesgen ^[2] ; Jörg,Höhfeld ^[10] ; Dieter O,Fürst ^[4] ; Bernd K,Fleischmann ^[4] ; Michael,Hesse ^[11]

作者单位： Institute of Physiology I, Life and Brain Center, Medical Faculty, University of Bonn, Bonn, Germany. ^[1] Central Institute for Engineering, Electronics and Analytics, ZEA-3, Forschungszentrum Jülich, Jülich, Germany. ^[2] Institute of Physiology II, University of Münster, Münster, Germany. ^[3] Department of Molecular Cell Biology, Institute for Cell Biology, University of Bonn, Bonn, Germany. ^[4] Institute of Experimental and Clinical Pharmacology and Toxicology, Division II, Faculty of Medicine, University of Freiburg, Freiburg, Germany. ^[5] Department of Anatomy and Embryology, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan. ^[6] Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA, USA. ^[7] Department of Cardiac Surgery, University of Bonn, Bonn, Germany. ^[8] German Sport University Cologne, Department of Molecular and Cellular Sport Medicine, Cologne, Germany. ^[9] CECAD, Medical Faculty and University Hospital, and Institute of Biochemistry, Faculty of Mathematics and Natural Sciences, University of Cologne, Cologne, Germany. ^[10] Institute of Physiology I, Life and Brain Center, Medical Faculty, University of Bonn, Bonn, Germany. bernd.fleischmann@uni-bonn.de. ^[11] Institute of Physiology I, Life and Brain Center, Medical Faculty, University of Bonn, Bonn, Germany. mhesse1@uni-bonn.de. ^[12]

主题词衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);动物(Animals);凋亡调节蛋白质类(Apoptosis Regulatory Proteins);自噬(Autophagy);结合部位(Binding Sites);心肌疾病(Cardiomyopathies);心肌病, 限制性(Cardiomyopathy, Restrictive);儿童(Child);疾病模型, 动物(Disease Models, Animal);基因表达调控(Gene Expression Regulation);心脏(Heart);热休克蛋白质类(Heat-Shock Proteins);人类(Humans);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);分子伴侣(Molecular Chaperones);突变(Mutation);蛋白质结合(Protein Binding);蛋白质组学(Proteomics);肌节(Sarcomeres)

DOI 10.1038/s41467-021-23858-7

PMID 34117258

发布时间 2021-06-30

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Overexpression of human BAG3<sup>P209L</sup> in mice causes restrictive cardiomyopathy.

Nature communications

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Nature communications

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Overexpression of human BAG3<sup>P209L</sup> in mice causes restrictive cardiomyopathy.

Nature communications

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Nature communications

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