Dehydroepiandrosterone alleviates intestinal inflammatory damage via GPR30-mediated Nrf2 activation and NLRP3 inflammasome inhibition in colitis mice.

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作者： Ji,Cao ^[1] ; Miaomiao,Lu ^[1] ; Weiyuan,Yan ^[1] ; Longlong,Li ^[1] ; Haitian,Ma ^[2]

作者单位： Key Laboratory of Animal Physiology and Biochemistry, College of Veterinary Medicine, Nanjing Agricultural University, Nanjing, 210095, PR China; MOE Joint International Research Laboratory of Animal Health and Food Safety, College of Veterinary Medicine, Nanjing Agricultural University, Nanjing, 210095, PR China. ^[1] Key Laboratory of Animal Physiology and Biochemistry, College of Veterinary Medicine, Nanjing Agricultural University, Nanjing, 210095, PR China; MOE Joint International Research Laboratory of Animal Health and Food Safety, College of Veterinary Medicine, Nanjing Agricultural University, Nanjing, 210095, PR China. Electronic address: mahaitian@njau.edu.cn. ^[2]

关键词 Colitis Dehydroepiandrosterone GPR30 NLRP3 inflammasome Nrf2

主题词动物(Animals);结肠炎(Colitis);去氢表雄酮(Dehydroepiandrosterone);炎性体(Inflammasomes);小鼠(Mice);NF-E2相关因子2(NF-E2-Related Factor 2);受体, G-蛋白偶联(Receptors, G-Protein-Coupled)

DOI 10.1016/j.freeradbiomed.2021.06.025

PMID 34182071

发布时间 2021-08-26

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