Glucose limitation activates AMPK coupled SENP1-Sirt3 signalling in mitochondria for T cell memory development.
第一作者:
Jianli,He
第一单位:
State Key Laboratory of Oncogenes and Related Genes, Renji Hospital Affiliated; Shanghai Key Laboratory for Tumor Microenvironment and Inflammation, Department of Biochemistry and Molecular Cell Biology, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
作者:
主题词
AMP活化蛋白激酶类(AMP-Activated Protein Kinases);乙酰化作用(Acetylation);动物(Animals);CD8阳性T淋巴细胞(CD8-Positive T-Lymphocytes);细胞系, 肿瘤(Cell Line, Tumor);细胞存活(Cell Survival);结肠肿瘤(Colonic Neoplasms);半胱氨酸内肽酶类(Cysteine Endopeptidases);果糖二磷酸盐类(Fructosediphosphates);GTP磷酸水解酶类(GTP Phosphohydrolases);葡萄糖(Glucose);免疫记忆(Immunologic Memory);代谢组学(Metabolomics);金属内肽酶类(Metalloendopeptidases);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);显微镜检查, 电子, 透射(Microscopy, Electron, Transmission);线粒体(Mitochondria);线粒体蛋白质类(Mitochondrial Proteins);氧化磷酸化(Oxidative Phosphorylation);抗衰老酶3(Sirtuin 3);SUMO化(Sumoylation);T淋巴细胞(T-Lymphocytes)
DOI
10.1038/s41467-021-24619-2
PMID
34272364
发布时间
2021-08-05
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