The Synaptic Vesicle Protein 2A Interacts With Key Pathogenic Factors in Alzheimer's Disease: Implications for Treatment.

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作者： Yanyan,Kong ^[1] ; Lin,Huang ^[2] ; Weihao,Li ^[2] ; Xuanting,Liu ^[2] ; Yinping,Zhou ^[2] ; Cuiping,Liu ^[2] ; Shibo,Zhang ^[2] ; Fang,Xie ^[1] ; Zhengwei,Zhang ^[1] ; Donglang,Jiang ^[1] ; Weiyan,Zhou ^[1] ; Ruiqing,Ni ^[3] ; Chencheng,Zhang ^[4] ; Bomin,Sun ^[4] ; Jiao,Wang ^[2] ; Yihui,Guan ^[1]

作者单位： PET Center, Huashan Hospital, Fudan University, Shanghai, China. ^[1] Laboratory of Molecular Neural Biology, School of Life Sciences, Shanghai University, Shanghai, China. ^[2] Institute for Biomedical Engineering, ETH Zurich, University of Zurich, Zurich, Switzerland. ^[3] Department of Neurosurgery, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China. ^[4]

关键词 Alzheimer’s disease Aβ PI3K signaling pathway Tau neurodegeneration synaptic vesicle protein 2A

DOI 10.3389/fcell.2021.609908

PMID 34277597

发布时间 2021-07-20

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Frontiers in cell and developmental biology

2021年9卷

609908页

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The Synaptic Vesicle Protein 2A Interacts With Key Pathogenic Factors in Alzheimer's Disease: Implications for Treatment.

Frontiers in cell and developmental biology

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Frontiers in cell and developmental biology

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The Synaptic Vesicle Protein 2A Interacts With Key Pathogenic Factors in Alzheimer's Disease: Implications for Treatment.

Frontiers in cell and developmental biology

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Frontiers in cell and developmental biology

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