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Selective activation of PFKL suppresses the phagocytic oxidative burst.

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第一作者： Neri,Amara

第一单位： Physiological Chemistry Department, Genentech, South San Francisco, CA 94080, USA.

作者： Neri,Amara ^[1] ; Madison P,Cooper ^[2] ; Maria A,Voronkova ^[2] ; Bradley A,Webb ^[2] ; Eric M,Lynch ^[3] ; Justin M,Kollman ^[3] ; Taylur,Ma ^[4] ; Kebing,Yu ^[4] ; Zijuan,Lai ^[5] ; Dewakar,Sangaraju ^[5] ; Nobuhiko,Kayagaki ^[1] ; Kim,Newton ^[1] ; Matthew,Bogyo ^[6] ; Steven T,Staben ^[7] ; Vishva M,Dixit ^[8]

作者单位： Physiological Chemistry Department, Genentech, South San Francisco, CA 94080, USA. ^[1] Biochemistry Department, West Virginia University, Morgantown, WV 26506, USA. ^[2] Biochemistry Department, University of Washington, Seattle, WA 98195, USA. ^[3] Microchemistry, Proteomics, and Lipidomics Department, Genentech, South San Francisco, CA 94080, USA. ^[4] Drug Metabolism and Pharmacokinetics Department, Genentech, South San Francisco, CA 94080, USA. ^[5] Pathology Department, Stanford University, Stanford, CA 94305, USA. ^[6] Discovery Chemistry Department, Genentech, South San Francisco, CA 94080, USA. ^[7] Physiological Chemistry Department, Genentech, South San Francisco, CA 94080, USA. Electronic address: dixit@gene.com. ^[8]

关键词 LDC7559 NA-11 NADPH NETosis NOX2 PFKL ROS neutrophils