ANP32B-mediated repression of p53 contributes to maintenance of normal and CML stem cells.

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第一作者： Shuo,Yang

第一单位： Department of Pathophysiology, State Key Laboratory of Oncogenes and Related Genes, Chinese Academy of Medical Sciences Research Unit 2019RU043, Ren-Ji Hospital, Shanghai Jiao Tong University School of Medicine (SJTU-SM), Shanghai, China.

作者： Shuo,Yang ^[1] ; Xiao-Na,Zhu ^[1] ; Hui-Lin,Zhang ^[2] ; Qian,Yang ^[2] ; Yu-Sheng,Wei ^[2] ; Di,Zhu ^[3] ; Meng-Di,Liu ^[4] ; Shao-Ming,Shen ^[2] ; Li,Xia ^[2] ; Ping,He ^[1] ; Meng-Kai,Ge ^[1] ; Yi-Lian,Pan ^[4] ; Meng,Zhao ^[5] ; Ying-Li,Wu ^[2] ; Jun-Ke,Zheng ^[2] ; Guo-Qiang,Chen ^[1] ; Yun,Yu ^[2]

作者单位： Department of Pathophysiology, State Key Laboratory of Oncogenes and Related Genes, Chinese Academy of Medical Sciences Research Unit 2019RU043, Ren-Ji Hospital, Shanghai Jiao Tong University School of Medicine (SJTU-SM), Shanghai, China. ^[1] Key Laboratory of Cell Differentiation and Apoptosis of Chinese Ministry of Education, Rui-Jin Hospital, SJTU-SM, Shanghai, China. ^[2] Department of Hematology, Ren-Ji Hospital, SJTU-SM, Shanghai, China. ^[3] International Peace Maternity and Child Health Hospital, SJTU-SM, Shanghai, China; and. ^[4] Key Laboratory of Stem Cells and Tissue Engineering, Zhongshan School of Medicine, Sun Yat-Sen University, Ministry of Education, Guangzhou, China. ^[5]