Flecainide Paradoxically Activates Cardiac Ryanodine Receptor Channels under Low Activity Conditions: A Potential Pro-Arrhythmic Action.

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作者： Samantha C,Salvage ^[1] ; Esther M,Gallant ^[2] ; James A,Fraser ^[3] ; Christopher L-H,Huang ^[1] ; Angela F,Dulhunty ^[1]

作者单位： Physiological Laboratory, University of Cambridge, Downing Street, Cambridge CB2 3EG, UK. ^[1] Department of Biochemistry, University of Cambridge, Tennis Court Road, Cambridge CB2 1QW, UK. ^[2] Eccles Institute of Neuroscience, John Curtin School of Medical Research, The Australian National University, 131 Garran Road, Acton 2601, Australia. ^[3]

关键词 RyR2 RyR2 activation RyR2 inhibition RyR2-P2328S atrial fibrillation catecholaminergic polymorphic ventricular tachycardia flecainide

主题词动物(Animals);抗心律失常药(Anti-Arrhythmia Agents);心律失常, 心性(Arrhythmias, Cardiac);钙(Calcium);氟卡尼(Flecainide);离子通道闸门(Ion Channel Gating);脂双层(Lipid Bilayers);膜电位(Membrane Potentials);小鼠(Mice);兰尼碱受体钙释放通道(Ryanodine Receptor Calcium Release Channel);肌浆网(Sarcoplasmic Reticulum)

DOI 10.3390/cells10082101

PMID 34440870

发布时间 2022-05-18

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Cells

2021年10卷8期

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Flecainide Paradoxically Activates Cardiac Ryanodine Receptor Channels under Low Activity Conditions: A Potential Pro-Arrhythmic Action.

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Flecainide Paradoxically Activates Cardiac Ryanodine Receptor Channels under Low Activity Conditions: A Potential Pro-Arrhythmic Action.

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