Selective FPR2 Agonism Promotes a Proresolution Macrophage Phenotype and Improves Cardiac Structure-Function Post Myocardial Infarction.
第一作者:
Ricardo A,García
第一单位:
Department of Cardiovascular and Fibrosis Drug Discovery, Bristol Myers Squibb, Princeton, New Jersey, USA.;Department of Medicine, University of California-San Diego, San Diego, California, USA.
作者:
关键词
BRET, bioluminescence resonance energy transferEC50, half maximal effective concentrationFPR2FPR2, formyl peptide receptor 2HFHF, heart failureI/R, ischemia-reperfusionIL, interleukinKO, knockoutLPS, lipopolysaccharideLV, left ventricle/ventricularMCP, monocyte chemoattractant proteinMIMI, myocardial infarctionSAA, serum amyloid ATNF, tumor necrosis factorWT, wild-typeformyl peptide receptor 2heart failuremRNA, messenger RNAmyocardial infarctionresolution
DOI
10.1016/j.jacbts.2021.07.007
PMID
34466754
发布时间
2021-09-03
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JACC. Basic to translational science
2021年6卷8期
676-689页
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