ACBD3 modulates KDEL receptor interaction with PKA for its trafficking via tubulovesicular carrier.

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作者： Xihua,Yue ^[1] ; Yi,Qian ^[1] ; Lianhui,Zhu ^[1] ; Bopil,Gim ^[2] ; Mengjing,Bao ^[1] ; Jie,Jia ^[1] ; Shuaiyang,Jing ^[3] ; Yijing,Wang ^[1] ; Chuanting,Tan ^[3] ; Francesca,Bottanelli ^[1] ; Pascal,Ziltener ^[3] ; Sunkyu,Choi ^[1] ; Piliang,Hao ^[3] ; Intaek,Lee ^[4]

作者单位： School of Life Science and Technology, ShanghaiTech University, Pudong, Shanghai, China. ^[1] School of Physical Science and Technology, ShanghaiTech University, Pudong, Shanghai, China. ^[2] University of Chinese Academy of Sciences, Beijing, China. ^[3] Institut für Biochemie, Freie Universität Berlin, Thielallee 63, 14195, Berlin, Germany. ^[4] Department of Cell Biology, Yale University School of Medicine, New Haven, CT, USA. ^[5] Proteomics Core, Weill Cornell Medicine-Qatar, Doha, Qatar. ^[6] School of Life Science and Technology, ShanghaiTech University, Pudong, Shanghai, China. Leeintaek@shanghaitech.edu.cn. ^[7] Shanghai Institute for Advanced Immunochemical Studies, Shanghai, China. Leeintaek@shanghaitech.edu.cn. ^[8]

关键词 ACBD3 Arf1-GTP ArfGAPs Golgi KDEL receptor Protein Kinase A

主题词衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);细胞膜(Cell Membrane);环AMP依赖性蛋白激酶类(Cyclic AMP-Dependent Protein Kinases);高尔基体(Golgi Apparatus);受体, 肽(Receptors, Peptide)

DOI 10.1186/s12915-021-01137-7

PMID 34493279

发布时间 2021-12-21

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准确一般字义偏差语意偏离译文作用小提交