Siglec-H-Deficient Mice Show Enhanced Type I IFN Responses, but Do Not Develop Autoimmunity After Influenza or LCMV Infections.

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作者： Nadine,Szumilas ^[1] ; Odilia B J,Corneth ^[2] ; Christian H K,Lehmann ^[3] ; Heike,Schmitt ^[4] ; Svenia,Cunz ^[5] ; Jolie G,Cullen ^[6] ; Talyn,Chu ^[1] ; Anita,Marosan ^[7] ; Attila,Mócsai ^[7] ; Vladimir,Benes ^[8] ; Dietmar,Zehn ^[9] ; Diana,Dudziak ^[10] ; Rudi W,Hendriks ^[7] ; Lars,Nitschke ^[3]

作者单位： Division of Genetics, Department of Biology, University of Erlangen-Nürnberg, Erlangen, Germany. ^[1] Department of Pulmonary Medicine, Erasmus MC, University Medical Center, Rotterdam, Netherlands. ^[2] Laboratory of Dendritic Cell Biology, Department of Dermatology, University Hospital Erlangen, Erlangen, Germany. ^[3] Deutsches Zentrum Immuntherapie (DZI), University Hospital Erlangen, University of Erlangen-Nürnberg, Erlangen, Germany. ^[4] Medical Immunology Campus Erlangen (MICE), University of Erlangen-Nürnberg, Erlangen, Germany. ^[5] First Department of Medicine, University Hospital Erlangen, Erlangen, Germany. ^[6] Division of Animal Physiology and Immunology, School of Life Sciences Weihenstephan, Technical University of Munich, Freising, Germany. ^[7] Department of Immune Modulation, University Hospital Erlangen, Erlangen, Germany. ^[8] Semmelweis University School of Medicine, Budapest, Hungary. ^[9] Genomics Core Facility, EMBL Heidelberg, Heidelberg, Germany. ^[10]

关键词 Interferon-alpha SLE Siglec TLR9 plasmacytoid dendritic cells