Hypophosphatasia: Vitamin B<sub>6</sub> status of affected children and adults.

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作者： Michael P,Whyte ^[1] ; Fan,Zhang ^[2] ; Deborah,Wenkert ^[3] ; Karen E,Mack ^[4] ; Vinieth N,Bijanki ^[5] ; Karen L,Ericson ^[6] ; Stephen P,Coburn ^[7]

作者单位： Center for Metabolic Bone Disease and Molecular Research, Shriners Hospitals for Children - St. Louis, St. Louis, MO 63110, USA; Division of Bone and Mineral Diseases, Department of Internal Medicine, Washington University School of Medicine at Barnes-Jewish Hospital, St. Louis, MO 63110, USA. Electronic address: mwhyte@shrinenet.org. ^[1] Center for Metabolic Bone Disease and Molecular Research, Shriners Hospitals for Children - St. Louis, St. Louis, MO 63110, USA. Electronic address: fzhang@shrinenet.org. ^[2] Center for Metabolic Bone Disease and Molecular Research, Shriners Hospitals for Children - St. Louis, St. Louis, MO 63110, USA. Electronic address: wenkert@i1.net. ^[3] Center for Metabolic Bone Disease and Molecular Research, Shriners Hospitals for Children - St. Louis, St. Louis, MO 63110, USA. Electronic address: kmack@shrinenet.org. ^[4] Center for Metabolic Bone Disease and Molecular Research, Shriners Hospitals for Children - St. Louis, St. Louis, MO 63110, USA. Electronic address: vbijanki@shrinenet.org. ^[5] Department of Chemistry, Purdue University Fort Wayne, Fort Wayne, IN 46805, USA. Electronic address: ericsonk@pfw.edu. ^[6] Department of Chemistry, Purdue University Fort Wayne, Fort Wayne, IN 46805, USA. Electronic address: Coburn@pfw.edu. ^[7]