IL-17C and IL-17RE Promote Wound Closure in a <i>Staphylococcus aureus</i>-Based Murine Wound Infection Model.

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作者： Linda,Pätzold ^[1] ; Alexandra,Stark ^[2] ; Felix,Ritzmann ^[3] ; Carola,Meier ^[4] ; Thomas,Tschernig ^[4] ; Jörg,Reichrath ^[2] ; Robert,Bals ^[3] ; Markus,Bischoff ^[1] ; Christoph,Beisswenger ^[3]

作者单位： Institute for Medical Microbiology and Hygiene, Saarland University, 66421 Homburg, Germany. ^[1] Department of Dermatology, Saarland University Hospital, 66421 Homburg, Germany. ^[2] Department of Internal Medicine V-Pulmonology, Allergology and Critical Care Medicine, Saarland University, 66421 Homburg, Germany. ^[3] Institute of Anatomy and Cell Biology, Saarland University, 66421 Homburg, Germany. ^[4]

关键词 Staphylococcus aureus interleukin-17C wound closure wound infection

DOI 10.3390/microorganisms9091821

PMID 34576717

发布时间 2021-10-01

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Microorganisms

2021年9卷9期

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IL-17C and IL-17RE Promote Wound Closure in a <i>Staphylococcus aureus</i>-Based Murine Wound Infection Model.

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IL-17C and IL-17RE Promote Wound Closure in a <i>Staphylococcus aureus</i>-Based Murine Wound Infection Model.

Microorganisms

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Microorganisms

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