Perinatal IL-1β-induced inflammation suppresses Tbr2<sup>+</sup> intermediate progenitor cell proliferation in the developing hippocampus accompanied by long-term behavioral deficits.

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作者： Stephanie,Veerasammy ^[1] ; Juliette,Van Steenwinckel ^[2] ; Tifenn,Le Charpentier ^[3] ; Joon Ho,Seo ^[2] ; Bobbi,Fleiss ^[3] ; Pierre,Gressens ^[1] ; Steven W,Levison ^[2]

作者单位： Department of Pharmacology, Physiology, and Neuroscience, Rutgers University, New Jersey Medical School, Cancer Center, 205 South Orange Avenue, Newark, NJ, 07103, USA. ^[1] Université de Paris, NeuroDiderot, Inserm, F-75019, Paris, France. ^[2] PremUP, F-75006, Paris, France. ^[3] School of Health and Biomedical Sciences, RMIT University, Bundoora, 3083, VIC, Australia. ^[4] Centre for the Developing Brain, Division of Imaging Sciences and Biomedical Engineering, King's College London, King's Health Partners, St. Thomas' Hospital, London, SE1 7EH, UK. ^[5]

关键词 Anxiety Autism Brain development Maternal immune activation Memory Neurogenesis Systemic inflammation

DOI 10.1016/j.bbih.2020.100106

PMID 34589867

发布时间 2023-11-07

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Brain, behavior, & immunity - health

2020年7卷

100106页

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Perinatal IL-1β-induced inflammation suppresses Tbr2<sup>+</sup> intermediate progenitor cell proliferation in the developing hippocampus accompanied by long-term behavioral deficits.

Brain, behavior, & immunity - health

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Brain, behavior, & immunity - health

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Perinatal IL-1β-induced inflammation suppresses Tbr2<sup>+</sup> intermediate progenitor cell proliferation in the developing hippocampus accompanied by long-term behavioral deficits.

Brain, behavior, & immunity - health

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Brain, behavior, & immunity - health

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