Early Chronic Memantine Treatment-Induced Transcriptomic Changes in Wild-Type and <i>Shank2</i>-Mutant Mice.

导出 LinkOut

翻译打印收藏纠错

作者： Ye-Eun,Yoo ^[1] ; Seungjoon,Lee ^[2] ; Woohyun,Kim ^[1] ; Hyosang,Kim ^[1] ; Changuk,Chung ^[1] ; Seungmin,Ha ^[2] ; Jinsu,Park ^[2] ; Yeonseung,Chung ^[3] ; Hyojin,Kang ^[3] ; Eunjoon,Kim ^[4]

作者单位： Department of Biological Sciences, Korea Advanced Institute of Science and Technology, Daejeon, South Korea. ^[1] Center for Synaptic Brain Dysfunctions, Institute for Basic Science, Daejeon, South Korea. ^[2] Department of Mathematical Sciences, Korea Advanced Institute of Science and Technology, Daejeon, South Korea. ^[3] Division of National Supercomputing, Korea Institute of Science and Technology Information, Daejeon, South Korea. ^[4]

关键词 NMDA receptor RNA-Seq Shank2 autism spectrum disorders memantine mitochondria ribosome synapse

DOI 10.3389/fnmol.2021.712576

PMID 34594187

发布时间 2021-10-02

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

Frontiers in molecular neuroscience

2021年14卷

712576页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Early Chronic Memantine Treatment-Induced Transcriptomic Changes in Wild-Type and <i>Shank2</i>-Mutant Mice.

Frontiers in molecular neuroscience

相似文献

Frontiers in molecular neuroscience

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Early Chronic Memantine Treatment-Induced Transcriptomic Changes in Wild-Type and <i>Shank2</i>-Mutant Mice.

Frontiers in molecular neuroscience

相似文献

Frontiers in molecular neuroscience

相关期刊

检索历史清除