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首页> Biochemical and biophysical research communications> IL-30 ameliorates imiquimod and K14-VEGF induced psoriasis-like disease by inhibiting both innate and adaptive immunity disorders.

IL-30 ameliorates imiquimod and K14-VEGF induced psoriasis-like disease by inhibiting both innate and adaptive immunity disorders.

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作者： Xiao,Liu ^[1] ; Zhonglan,Hu ^[2] ; Jun,Zhang ^[3] ; Teng,Ma ^[3] ; Wenlin,Wu ^[3] ; Xiaoqiong,Wei ^[3] ; Zhen,Wang ^[3] ; Huaping,Zhen ^[3] ; Hong,Zhou ^[3] ; Nongyu,Huang ^[3] ; Jiong,Li ^[4]

作者单位： Human Sperm Bank, Key Laboratory of Birth Defects and Related Diseases of Women and Children of MOE, State Key Laboratory of Biotherapy, West China Second Hospital, Sichuan University, Chengdu, 610041, Sichuan, China. ^[1] Department of Clinical Laboratory, The First Affiliated Hospital of Zhengzhou University, Zhengzhou, 450003, China. ^[2] State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, And Collaborative Innovation Center for Biotherapy, Chengdu, 610041, China. ^[3] State Key Laboratory of Biotherapy and Cancer Center, West China Hospital, Sichuan University, And Collaborative Innovation Center for Biotherapy, Chengdu, 610041, China. Electronic address: lijionghh@scu.edu.cn. ^[4]

关键词 Adaptive immunity IL-30 IMQ Innate immunity Krt14-Vegfa Psoriasis

主题词适应性免疫(Adaptive Immunity);动物(Animals);细胞系(Cell Line);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);细胞因子类(Cytokines);人类(Humans);炎症(Inflammation);白细胞介素类(Interleukins);角蛋白14(Keratin-14);角蛋白细胞(Keratinocytes);淋巴细胞(Lymphocytes);小鼠(Mice);银屑病(Psoriasis);信号传导(Signal Transduction);血管内皮生长因子A(Vascular Endothelial Growth Factor A)

DOI 10.1016/j.bbrc.2021.09.042

PMID 34597998

发布时间 2021-12-27

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Biochemical and biophysical research communications

97-104页

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