circFAM160A2 Promotes Mitochondrial Stabilization and Apoptosis Reduction in Osteoarthritis Chondrocytes by Targeting miR-505-3p and SIRT3.

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作者： Jiapeng,Bao ^[1] ; Changjian,Lin ^[2] ; Xing,Zhou ^[3] ; Diana,Ma ^[1] ; Lujie,Ge ^[2] ; Kai,Xu ^[3] ; Safwat Adel Abdo,Moqbel ^[1] ; Yuzhe,He ^[2] ; Chiyuan,Ma ^[3] ; Jisheng,Ran ^[1] ; Lidong,Wu ^[2]

作者单位： Department of Orthopedic Surgery, The Second Affiliated Hospital, Zhejiang University School of Medicine, Hangzhou City, Zhejiang Province, China. ^[1] Orthopedics Research Institute of Zhejiang University, Hangzhou City, Zhejiang Province, China. ^[2] Key Laboratory of Motor System Disease Research and Precision Therapy of Zhejiang Province, Hangzhou City, Zhejiang Province, China. ^[3]

主题词细胞凋亡(Apoptosis);细胞增殖(Cell Proliferation);软骨细胞(Chondrocytes);人类(Humans);炎症(Inflammation);微RNAs(MicroRNAs);线粒体(Mitochondria);骨关节炎(Osteoarthritis);抗衰老酶3(Sirtuin 3)

DOI 10.1155/2021/5712280

PMID 34646424

发布时间 2024-04-03

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Oxidative medicine and cellular longevity

2021年2021卷

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circFAM160A2 Promotes Mitochondrial Stabilization and Apoptosis Reduction in Osteoarthritis Chondrocytes by Targeting miR-505-3p and SIRT3.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

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circFAM160A2 Promotes Mitochondrial Stabilization and Apoptosis Reduction in Osteoarthritis Chondrocytes by Targeting miR-505-3p and SIRT3.

Oxidative medicine and cellular longevity

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Oxidative medicine and cellular longevity

相关期刊

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