换一批
换一批Bisphosphonate-enoxacin inhibit osteoclast formation and function by abrogating RANKL-induced JNK signalling pathways during osteoporosis treatment.
作者：
主题词
肌动蛋白类(Actins);动物(Animals);骨质吸收(Bone Resorption);骨和骨组织(Bone and Bones);二膦酸盐类(Diphosphonates);疾病模型, 动物(Disease Models, Animal);疾病易感性(Disease Susceptibility);依诺沙星(Enoxacin);基因表达调控(Gene Expression Regulation);MAP激酶信号系统(MAP Kinase Signaling System);小鼠(Mice);破骨细胞(Osteoclasts);骨生成(Osteogenesis);骨质疏松(Osteoporosis);RANK配体(RANK Ligand);治疗结果(Treatment Outcome);X线显微体层摄影术(X-Ray Microtomography)
DOI
10.1111/jcmm.16949
PMID
34651433
发布时间
2022-03-16
- 浏览0
Journal of cellular and molecular medicine
10126-10139页
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