The neuroprotection of hyperbaric oxygen therapy against traumatic brain injury via NF-κB/MAPKs-CXCL1 signaling pathways.

导出 LinkOut

翻译打印收藏纠错

作者： Anqi,Xia ^[1] ; Huan,Huang ^[2] ; Wenjun,You ^[1] ; Ying,Liu ^[2] ; Hongqin,Wu ^[3] ; Su,Liu ^[4]

作者单位： Department of Rehabilitation Medicine, Affiliated Hospital of Nantong University, Nantong, 226001, Jiangsu, China. ^[1] School of Medicine, Nantong University, Nantong, 226001, Jiangsu, China. ^[2] Department of Geriatrics, the Second Peoples Hospital of Nantong, Affiliated of Nantong University, Nantong, 226001, Jiangsu, China. ^[3] Department of Pathology, Affiliated Hospital of Nantong University, Nantong, China. ^[4] Department of Rehabilitation Medicine, Affiliated Hospital of Nantong University, Nantong, 226001, Jiangsu, China. 327202278@qq.com. ^[5]

关键词 CXCL1 CXCR2 Hyperbaric oxygen Traumatic brain injury

主题词动物(Animals);趋化因子CXCL1(Chemokine CXCL1);高压氧(Hyperbaric Oxygenation);MAP激酶信号系统(MAP Kinase Signaling System);NF-κB(NF-kappa B);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley)

DOI 10.1007/s00221-021-06249-8

PMID 34687331

发布时间 2022-02-02

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交