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Metformin promotes anticancer activity of NK cells in a p38 MAPK dependent manner.

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第一作者： Wenjiao,Xia

第一单位： Key Laboratory of Marine Drugs, Chinese Ministry of Education, School of Medicine and Pharmacy, Ocean University of China, Qingdao, P. R. China.

作者： Wenjiao,Xia ^[1] ; Xin,Qi ^[1] ; Mingfeng,Li ^[1] ; Yu,Wu ^[1] ; Lulu,Sun ^[1] ; Xinglong,Fan ^[2] ; Yuan,Yuan ^[3] ; Jing,Li ^[4]

作者单位： Key Laboratory of Marine Drugs, Chinese Ministry of Education, School of Medicine and Pharmacy, Ocean University of China, Qingdao, P. R. China. ^[1] Department of Thoracic Surgery, Qilu Hospital (Qingdao), Cheeloo College of Medicine, Shandong University, Qingdao, China. ^[2] Department of Laboratory, Yushan Campus Hospital, Hospital of Ocean University of China, Qingdao, P. R. China. ^[3] Key Laboratory of Marine Drugs, Chinese Ministry of Education, School of Medicine and Pharmacy, Ocean University of China, Qingdao, P. R. China.;Open Studio for Druggability Research of Marine Natural Products, Pilot National Laboratory for Marine Science and Technology (Qingdao), Laboratory for Marine Drugs and Bioproducts of Qingdao National Laboratory for Marine Science and Technology, Qingdao, P. R. China. ^[4]

关键词 Metformin NK cells cancer surveillance immunotherapy p38 MAPK

医学主题词动物(Animals);糖尿病, 2型(Diabetes Mellitus, Type 2);杀伤细胞, 天然(Killer Cells, Natural);黑色素瘤(Melanoma);二甲双胍(Metformin);小鼠(Mice);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)

DOI 10.1080/2162402X.2021.1995999

PMID 34745769

发布时间 2022-01-03

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