Hippocampal CysLT1R overexpression or activation accelerates memory deficits, synaptic dysfunction, and amyloidogenesis in young APP/PS1 transgenic mice.

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作者： Shun-Chang,Fang ^[1] ; Jun-Jie,Wang ^[1] ; Fang,Chen ^[1] ; Su-Su,Tang ^[1] ; Rong-Hao,Mu ^[1] ; Dan-Hua,Yuan ^[1] ; Jia-Jia,Zhao ^[1] ; Hao,Hong ^[1] ; Yan,Long ^[1]

作者单位： Department of Pharmacology, Key Laboratory of Neuropsychiatric Diseases, China Pharmaceutical University, Nanjing, China. ^[1]

关键词 Alzheimer’s disease (AD)Cysteinyl leukotrienes receptor 1 (CysLT1R)amyloidogenesis memory synaptic plasticity

DOI 10.21037/atm-21-4518

PMID 34790737

发布时间 2022-04-28

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Annals of translational medicine

2021年9卷20期

1531页

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Hippocampal CysLT1R overexpression or activation accelerates memory deficits, synaptic dysfunction, and amyloidogenesis in young APP/PS1 transgenic mice.

Annals of translational medicine

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Annals of translational medicine

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Hippocampal CysLT1R overexpression or activation accelerates memory deficits, synaptic dysfunction, and amyloidogenesis in young APP/PS1 transgenic mice.

Annals of translational medicine

相似文献

Annals of translational medicine

相关期刊

检索历史清除