Iron Metabolism as a Potential Mechanism for Inducing TRAIL-Mediated Extrinsic Apoptosis Using Methylsulfonylmethane in Embryonic Cancer Stem Cells.

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作者： Nipin,Sp ^[1] ; Dong Young,Kang ^[1] ; Eun Seong,Jo ^[2] ; Jin-Moo,Lee ^[2] ; Kyoung-Jin,Jang ^[3]

作者单位： Department of Pathology, Institute of Biomedical Science and Technology, School of Medicine, Konkuk University, Chungju 27478, Korea. ^[1] Pharmacological Research Division, National Institute of Food and Drug Safety Evaluation, Osong Health Technology Administration Complex, Cheongju-si 28159, Korea. ^[2] SK Bioscience, Seongnam-si 13493, Korea. ^[3]

关键词 MSM NCCIT NTERA-2 TRAIL iron metabolism miR-130a/miR-221/miR-222 p38/p53/ERK signaling

主题词细胞凋亡(Apoptosis);DNA损伤(DNA Damage);二甲亚砜(Dimethyl Sulfoxide);胚胎性癌干细胞(Embryonal Carcinoma Stem Cells);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);铁(Iron);微RNAs(MicroRNAs);模型, 生物学(Models, Biological);活性氧(Reactive Oxygen Species);砜类(Sulfones);TNF相关凋亡诱导配体(TNF-Related Apoptosis-Inducing Ligand);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)

DOI 10.3390/cells10112847

PMID 34831070

发布时间 2022-01-04

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2021年10卷11期

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Iron Metabolism as a Potential Mechanism for Inducing TRAIL-Mediated Extrinsic Apoptosis Using Methylsulfonylmethane in Embryonic Cancer Stem Cells.

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Iron Metabolism as a Potential Mechanism for Inducing TRAIL-Mediated Extrinsic Apoptosis Using Methylsulfonylmethane in Embryonic Cancer Stem Cells.

Cells

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