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Dysfunction of the circadian clock in the kidney tubule leads to enhanced kidney gluconeogenesis and exacerbated hyperglycemia in diabetes.

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第一作者： Camille,Ansermet

第一单位： Department of Biomedical Sciences, University of Lausanne, Lausanne, Switzerland.

作者： Camille,Ansermet ^[1] ; Gabriel,Centeno ^[1] ; Yohan,Bignon ^[1] ; Daniel,Ortiz ^[2] ; Sylvain,Pradervand ^[3] ; Andy,Garcia ^[1] ; Laure,Menin ^[2] ; Frédéric,Gachon ^[4] ; Hikari Ai,Yoshihara ^[5] ; Dmitri,Firsov ^[6]

作者单位： Department of Biomedical Sciences, University of Lausanne, Lausanne, Switzerland. ^[1] Mass Spectrometry Service, Institute of Chemical Sciences and Engineering, Ecole Polytechnique Fédérale de Lausanne, Lausanne, Switzerland. ^[2] Genomic Technologies Facility, University of Lausanne, Lausanne, Switzerland. ^[3] Department of Biomedical Sciences, University of Lausanne, Lausanne, Switzerland; Institute for Molecular Bioscience, The University of Queensland, Queensland, Australia. ^[4] Institute of Physics, Ecole Polytechnique Fédérale de Lausanne, Lausanne, Switzerland. Electronic address: hikari.yoshihara@epflu.ch. ^[5] Department of Biomedical Sciences, University of Lausanne, Lausanne, Switzerland. Electronic address: dmitri.firsov@unil.ch. ^[6]

关键词 circadian clock diabetes gluconeogenesis proximal tubule