Redefining the biological basis of lineage-ambiguous leukemia through genomics: BCL11B deregulation in acute leukemias of ambiguous lineage.

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作者： Lindsey E,Montefiori ^[1] ; Charles G,Mullighan ^[2]

作者单位： Department of Pathology, St. Jude Children's Research Hospital, Memphis, TN, USA. ^[1] Department of Pathology, St. Jude Children's Research Hospital, Memphis, TN, USA. Electronic address: charles.mullighan@stjude.org. ^[2]

关键词 14q32 ALAL BCL11B Enhancer hijacking Lineage ambiguous Mixed phenotype acute leukemia

主题词儿童(Child);基因组学(Genomics);人类(Humans);免疫表型分型(Immunophenotyping);前体细胞淋巴母细胞白血病淋巴瘤(Precursor Cell Lymphoblastic Leukemia-Lymphoma);前瞻性研究(Prospective Studies);阻遏蛋白质类(Repressor Proteins);肿瘤抑制蛋白质类(Tumor Suppressor Proteins)

DOI 10.1016/j.beha.2021.101329

PMID 34865701

发布时间 2022-12-02

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Best practice & research. Clinical haematology

2021年34卷4期

101329页

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Redefining the biological basis of lineage-ambiguous leukemia through genomics: BCL11B deregulation in acute leukemias of ambiguous lineage.

Best practice & research. Clinical haematology

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Redefining the biological basis of lineage-ambiguous leukemia through genomics: BCL11B deregulation in acute leukemias of ambiguous lineage.

Best practice & research. Clinical haematology

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Best practice & research. Clinical haematology

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