Autophagy-Induced HDAC6 Activity During Hypoxia Regulates Mitochondrial Energy Metabolism Through the β-Catenin/COUP-TFII Axis in Hepatocellular Carcinoma Cells.

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第一作者： Xiaoyu,Yan

第一单位： Key Laboratory of Pathobiology, Ministry of Education, Department of Pathophysiology, College of Basic Medical Sciences, Jilin University, Changchun, China.

作者： Xiaoyu,Yan ^[1] ; Xianzhi,Qu ^[2] ; Buhan,Liu ^[1] ; Yuanxin,Zhao ^[1] ; Long,Xu ^[1] ; Sihang,Yu ^[1] ; Jian,Wang ^[1] ; Liying,Wang ^[3] ; Jing,Su ^[1]

作者单位： Key Laboratory of Pathobiology, Ministry of Education, Department of Pathophysiology, College of Basic Medical Sciences, Jilin University, Changchun, China. ^[1] Department of Hepatobiliary & Pancreatic Surgery, The Second Hospital of Jilin University, Jilin University, Changchun, China. ^[2] Department of Molecular Biology, College of Basic Medical Sciences, Norman Bethune Health Science Center, Jilin University, Changchun, China. ^[3]

关键词 HDAC6 autophagy hepatocellular carcinoma hypoxia mitochondrial energy metabolism β-catenin

DOI 10.3389/fonc.2021.742460

PMID 34868939

发布时间 2021-12-07

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Frontiers in oncology

2021年11卷

742460页

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Autophagy-Induced HDAC6 Activity During Hypoxia Regulates Mitochondrial Energy Metabolism Through the β-Catenin/COUP-TFII Axis in Hepatocellular Carcinoma Cells.

Frontiers in oncology

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Frontiers in oncology

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Autophagy-Induced HDAC6 Activity During Hypoxia Regulates Mitochondrial Energy Metabolism Through the β-Catenin/COUP-TFII Axis in Hepatocellular Carcinoma Cells.

Frontiers in oncology

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Frontiers in oncology

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