Steroid-sensitive nephrotic syndrome candidate gene CLVS1 regulates podocyte oxidative stress and endocytosis.

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作者： Brandon M,Lane ^[1] ; Megan,Chryst-Stangl ^[1] ; Guanghong,Wu ^[1] ; Mohamed,Shalaby ^[2] ; Sherif,El Desoky ^[2] ; Claire C,Middleton ^[1] ; Kinsie,Huggins ^[1] ; Amika,Sood ^[3] ; Alejandro,Ochoa ^[3] ; Andrew F,Malone ^[4] ; Ricardo,Vancini ^[5] ; Sara E,Miller ^[5] ; Gentzon,Hall ^[6] ; So Young,Kim ^[7] ; David N,Howell ^[5] ; Jameela A,Kari ^[2] ; Rasheed,Gbadegesin ^[6]

作者单位： Department of Pediatrics, Division of Nephrology, and Duke Molecular Physiology Institute, Duke University School of Medicine, Durham, North Carolina, USA. ^[1] Pediatric Department, Pediatric Nephrology Center of Excellence, King Abdulaziz University, Jeddah, Saudi Arabia. ^[2] Department of Biostatistics and Bioinformatics and Duke Center for Statistical Genetics and Genomics, Duke University, Durham, North Carolina, USA. ^[3] Department of Medicine, Division of Nephrology, Washington University School of Medicine in St. Louis, St. Louis, Missouri, USA. ^[4] Department of Pathology. ^[5] Department of Pediatrics, Division of Nephrology, and Duke Molecular Physiology Institute, Duke University School of Medicine, Durham, North Carolina, USA.;Department of Medicine, Division of Nephrology; and. ^[6] Department of Molecular Genetics and Microbiology, Duke University School of Medicine, Durham, North Carolina, USA. ^[7]

关键词 Chronic kidney disease Genetic diseases Monogenic diseases Nephrology

主题词肾上腺皮质激素类(Adrenal Cortex Hormones);动物(Animals);细胞凋亡(Apoptosis);载体蛋白质类(Carrier Proteins);细胞, 培养的(Cells, Cultured);胞吞作用(Endocytosis);基因敲除技术(Gene Knockout Techniques);遗传关联研究(Genetic Association Studies);高通量核苷酸序列分析(High-Throughput Nucleotide Sequencing);人类(Humans);肾病综合征(Nephrotic Syndrome);氧化性应激(Oxidative Stress);足细胞(Podocytes);活性氧(Reactive Oxygen Species);斑马鱼(Zebrafish);斑马鱼蛋白质类(Zebrafish Proteins)

DOI 10.1172/jci.insight.152102

PMID 34874915

发布时间 2024-09-23

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Steroid-sensitive nephrotic syndrome candidate gene CLVS1 regulates podocyte oxidative stress and endocytosis.

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Steroid-sensitive nephrotic syndrome candidate gene CLVS1 regulates podocyte oxidative stress and endocytosis.

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