Oxidative stress-induced FABP5 S-glutathionylation protects against acute lung injury by suppressing inflammation in macrophages.

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作者： Yuxian,Guo ^[1] ; Yaru,Liu ^[2] ; Shihao,Zhao ^[1] ; Wangting,Xu ^[1] ; Yiqing,Li ^[1] ; Pengwei,Zhao ^[1] ; Di,Wang ^[3] ; Hongqiang,Cheng ^[2] ; Yuehai,Ke ^[1] ; Xue,Zhang ^[4]

作者单位： Department of Pathology and Pathophysiology and Sir Run Run Shaw Hospital, Zhejiang University School of Medicine, 310058, Hangzhou, P.R. China. ^[1] Institute of Immunology, Zhejiang University School of Medicine, 310058, Hangzhou, P.R. China. ^[2] Department of Biochemistry, Zhejiang University School of Medicine, 310058, Hangzhou, P.R. China. ^[3] Department of Cardiology of Second Affiliated Hospital, Zhejiang University School of Medicine, 310058, Hangzhou, P.R. China. ^[4] Department of Pathology and Pathophysiology and Sir Run Run Shaw Hospital, Zhejiang University School of Medicine, 310058, Hangzhou, P.R. China. yke@zju.edu.cn. ^[5] Department of Respiratory Medicine of Sir Run Run Shaw Hospital, Zhejiang University School of Medicine, 310058, Hangzhou, P.R. China. yke@zju.edu.cn. ^[6] Zhejiang Laboratory for Systems and Precision Medicine, Zhejiang University Medical Center, 311121, Hangzhou, China. yke@zju.edu.cn. ^[7] Department of Pathology and Pathophysiology and Sir Run Run Shaw Hospital, Zhejiang University School of Medicine, 310058, Hangzhou, P.R. China. zhangxue@zju.edu.cn. ^[8] Department of Respiratory Medicine of Sir Run Run Shaw Hospital, Zhejiang University School of Medicine, 310058, Hangzhou, P.R. China. zhangxue@zju.edu.cn. ^[9]