Transcriptomics of single dose and repeated carbon black and ozone inhalation co-exposure highlight progressive pulmonary mitochondrial dysfunction.

导出 OA

翻译打印收藏纠错

作者： Quincy A,Hathaway ^[1] ; Nairrita,Majumder ^[2] ; William T,Goldsmith ^[3] ; Amina,Kunovac ^[3] ; Mark V,Pinti ^[4] ; Jack R,Harkema ^[3] ; Vince,Castranova ^[4] ; John M,Hollander ^[1] ; Salik,Hussain ^[2]

作者单位： Division of Exercise Physiology, West Virginia University School of Medicine, Morgantown, WV, USA. ^[1] Mitochondria, Metabolism and Bioenergetics Working Group, West Virginia University School of Medicine, Morgantown, WV, USA. ^[2] Center for Inhalation Toxicology (iTOX), West Virginia University School of Medicine, Morgantown, WV, USA. ^[3] Department of Physiology and Pharmacology, West Virginia University School of Medicine, 64 Medical Center Drive, PO Box 9229, Morgantown, WV, 26506-9229, USA. ^[4] West Virginia University School of Pharmacy, Morgantown, WV, USA. ^[5] Department of Pathobiology and Diagnostic Investigation, College of Veterinary Medicine, Michigan State University, East Lansing, MI, USA. ^[6] Mitochondria, Metabolism and Bioenergetics Working Group, West Virginia University School of Medicine, Morgantown, WV, USA. salik.hussain@hsc.wvu.edu. ^[7] Center for Inhalation Toxicology (iTOX), West Virginia University School of Medicine, Morgantown, WV, USA. salik.hussain@hsc.wvu.edu. ^[8] Department of Physiology and Pharmacology, West Virginia University School of Medicine, 64 Medical Center Drive, PO Box 9229, Morgantown, WV, 26506-9229, USA. salik.hussain@hsc.wvu.edu. ^[9]

关键词 Carbon black Environmental exposure Genomics Inhalation toxicology Lung Mitochondria Ozone Ultrafine/nanoparticle