ERK1/2 inhibition promotes robust myotube growth via CaMKII activation resulting in myoblast-to-myotube fusion.-论文-万方医学网

第一作者： Tamar,Eigler

第一单位： Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel.

作者： Tamar,Eigler ^[1] ; Giulia,Zarfati ^[2] ; Emmanuel,Amzallag ^[1] ; Sansrity,Sinha ^[2] ; Nadav,Segev ^[2] ; Yishaia,Zabary ^[3] ; Assaf,Zaritsky ^[3] ; Avraham,Shakked ^[1] ; Kfir-Baruch,Umansky ^[1] ; Eyal D,Schejter ^[4] ; Douglas P,Millay ^[5] ; Eldad,Tzahor ^[6] ; Ori,Avinoam ^[7]

作者单位： Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel. ^[1] Department of Biomolecular Sciences, Weizmann Institute of Science, Rehovot, Israel. ^[2] Department of Software & Information Systems Engineering, Ben Gurion University, Be'er Sheva, Israel. ^[3] Department of Molecular Genetics, Weizmann Institute of Science, Rehovot, Israel. ^[4] Division of Molecular Cardiovascular Biology, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, USA; Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, OH, USA. ^[5] Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot, Israel. Electronic address: eldad.tzahor@weizmann.ac.il. ^[6] Department of Biomolecular Sciences, Weizmann Institute of Science, Rehovot, Israel. Electronic address: ori.avinoam@weizmann.ac.il. ^[7]

主题词肌动蛋白类(Actins);动物(Animals);钙(Calcium);钙-钙调素依赖性蛋白激酶2型(Calcium-Calmodulin-Dependent Protein Kinase Type 2);细胞分化(Cell Differentiation);细胞融合(Cell Fusion);细胞增殖(Cell Proliferation);酶激活(Enzyme Activation);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);小鼠, 近交C57BL(Mice, Inbred C57BL);模型, 生物学(Models, Biological);肌纤维, 骨骼(Muscle Fibers, Skeletal);肌蛋白质类(Muscle Proteins);成肌细胞(Myoblasts);蛋白质结合(Protein Binding);蛋白激酶抑制剂(Protein Kinase Inhibitors);受体, 维甲酸(Receptors, Retinoic Acid);信号传导(Signal Transduction);rac1 GTP结合蛋白质(rac1 GTP-Binding Protein);小鼠(Mice)

DOI 10.1016/j.devcel.2021.11.022

PMID 34932950

发布时间 2024-02-26

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ERK1/2 inhibition promotes robust myotube growth via CaMKII activation resulting in myoblast-to-myotube fusion.

Developmental cell

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ERK1/2 inhibition promotes robust myotube growth via CaMKII activation resulting in myoblast-to-myotube fusion.

Developmental cell

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Developmental cell

相关期刊

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