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首页> PLoS genetics> Co-existing TP53 and ARID1A mutations promote aggressive endometrial tumorigenesis.

Co-existing TP53 and ARID1A mutations promote aggressive endometrial tumorigenesis.

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作者： Jake J,Reske ^[1] ; Mike R,Wilson ^[1] ; Jeanne,Holladay ^[1] ; Rebecca A,Siwicki ^[2] ; Hilary,Skalski ^[1] ; Shannon,Harkins ^[1] ; Marie,Adams ^[2] ; John I,Risinger ^[1] ; Galen,Hostetter ^[3] ; Ken,Lin ^[4] ; Ronald L,Chandler ^[5]

作者单位： Department of Obstetrics, Gynecology and Reproductive Biology, College of Human Medicine, Michigan State University, Grand Rapids, Michigan, United States of America. ^[1] Genomics Core Facility, Van Andel Research Institute, Grand Rapids, Michigan, United States of America. ^[2] Department of Women's Health, Spectrum Health System, Grand Rapids, Michigan, United States of America. ^[3] Pathology and Biorepository Core, Van Andel Research Institute, Grand Rapids, Michigan, United States of America. ^[4] Department of Obstetrics & Gynecology and Women's Health, Albert Einstein College of Medicine, Bronx, New York, New York, United States of America. ^[5] Department of Epigenetics, Van Andel Research Institute, Grand Rapids, Michigan, United States of America. ^[6]

主题词腺癌(Adenocarcinoma);动物(Animals);原位癌(Carcinoma in Situ);I类磷脂酰肌醇3-激酶类(Class I Phosphatidylinositol 3-Kinases);DNA结合蛋白质类(DNA-Binding Proteins);子宫内膜增生(Endometrial Hyperplasia);子宫内膜肿瘤(Endometrial Neoplasms);子宫内膜(Endometrium);上皮-间质转化(Epithelial-Mesenchymal Transition);女(雌)性(Female);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);小鼠(Mice);突变(Mutation);转录因子(Transcription Factors);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53);肿瘤抑制蛋白质类(Tumor Suppressor Proteins)

DOI 10.1371/journal.pgen.1009986

PMID 34941867

发布时间 2022-02-16

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PLoS genetics

PLoS genetics

2021年17卷12期

e1009986页

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