Formimidoyltransferase cyclodeaminase prevents the starvation-induced liver hepatomegaly and dysfunction through downregulating mTORC1.

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作者： Wenfeng,Zhang ^[1] ; Chaoying,Wu ^[2] ; Rui,Ni ^[1] ; Qifen,Yang ^[1] ; Lingfei,Luo ^[1] ; Jianbo,He ^[1]

作者单位： Institute of Developmental Biology and Regenerative Medicine, Southwest University, Beibei, Chongqing, China. ^[1] Chongqing Key Laboratory of Oral Diseases and Biomedical Sciences, Yubei, Chongqing, China. ^[2]

主题词脱氨酶类(Ammonia-Lyases);动物(Animals);疾病模型, 动物(Disease Models, Animal);谷氨酸亚胺甲基转移酶(Glutamate Formimidoyltransferase);肝细胞(Hepatocytes);肝肿大(Hepatomegaly);人类(Humans);肝(Liver);多蛋白复合物(Multiprotein Complexes);突变(Mutation);磷酰化(Phosphorylation);核糖体蛋白质S6(Ribosomal Protein S6);信号传导(Signal Transduction);绝食(Starvation);斑马鱼(Zebrafish)

DOI 10.1371/journal.pgen.1009980

PMID 34941873

发布时间 2022-02-16

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PLoS genetics

2021年17卷12期

e1009980页

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Formimidoyltransferase cyclodeaminase prevents the starvation-induced liver hepatomegaly and dysfunction through downregulating mTORC1.

PLoS genetics

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PLoS genetics

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Formimidoyltransferase cyclodeaminase prevents the starvation-induced liver hepatomegaly and dysfunction through downregulating mTORC1.

PLoS genetics

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PLoS genetics

相关期刊

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